sitting, crawling and walking are delayed

loss of strength and muscle mass

ataxic gait

both decreased and increased deep tendon reflexes

progressive spasticity

progressive paraplegia with loss of ability to walk, due to calcification of the ligamenta flava and congenital stenosis (narrowing) of the spinal canal

sensory integration issues and sensory defensiveness, most noticeable in early childhood

"stimulus-induced drop episodes (SIDEs)", unexpected tactile or auditory stimuli or excitement triggers a brief collapse but no loss of consciousness. Treatment includes medications such as valporate, clonazepam, selective serotonin uptake inhibitors or limotrigine.

	affects approximately 20% of patients Stephenson et all (2005) recorded a prevalence of 20% (34/170) from the CLS Foundation database. Stephenson et all (2005) have also emphasized that the nature of the movement disorder may change with age and that a single individual may have more than one type of neurologic sign, ranging from cataplexy that varies with stimulus, hyperekplexia, a prolonged tonic reaction, and true epileptic seizures.
	Epileptic seizures affect about 5% of individuals [Stephenson et all 2005]
	unexpected tactile or auditory stimuli/excitement trigger a 60- to 80-millisecond electromyographic (EMG) silence in the lower limbs that results in a brief collapse though no loss of consciousness [Crow et all 1998, Nakamura et all 1998].
	frequency of attacks may cause the need for a wheelchair to prevent injury
	individuals with persistent drop episodes may be at higher risk for spinal cord compression injuries.